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A highly successful model from other cancer types involves the multistep carcinogenesis sequence in which there is a morphological spectrum of cellular/nuclear abnormalities that progress over time, ranging from normal, to mild atypia/dysplasia to severe atypia/carcinoma in situ (e.g., intraepithelial neoplasia ), before the development of invasive cancers with metastatic potential ( O’Shaughnessy et al. Given this unsatisfactory state of early detection of established prostate cancer using serum PSA testing/screening, there is great interest in developing strategies to prevent prostate cancer altogether. Furthermore, many men with high-risk localized disease are not offered curative procedures and may be undertreated ( Silberstein et al. First, the radical treatments mentioned above are often inadequate because a significant fraction of those with intermediate- or high-risk clinically localized prostate cancer treated with curative intent manner experience cancer recurrence and disease-specific death ( Silberstein et al. In addition to potential overtreatment of low-grade/low-risk cancers, there are still significant problems encountered for men with intermediate and high-risk clinically localized prostate cancer (Gleason score 7 or higher). Over the last several years, an increasing use of active surveillance and watchful waiting appears to be mitigating this overtreatment problem, at least somewhat ( Tosoian et al. And, these treatments in men with only Gleason score 6 tumors are now considered to represent overtreatment ( Loeb et al. 5 Yet, the majority of these men in the recent past have nevertheless been treated with aggressive regimens, which may result in significant side effects. 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